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Sсiеncе 2016 №6273 February 05
Washington, DC: AAAS, 2016. — 117 p. — ISSN 0036-8075
Science is the world's leading joual of original scientific research, global news, and commentary.
It the academic joual of the American Association for the Advancement of Science and is considered one of the world's most prestigious scientific jouals. The major focus of the joual is publishing important original scientific research and research reviews, but Science also publishes science-related news, opinions on science policy and other matters of interest to scientists and others who are conceed with the wide implications of science and technology. Although most scientific jouals focus on a specific field, Science and its rival Nature cover the full range of scientific disciplines.
From the contents
Perspectives
The mite that jumped, the bee that traveled, the disease that followed
Throttling back the heart’s molecular motor
Addressing an antiferromagnetic memory
Aging, alopecia, and stem cells
Nanoparticles meet their sticky ends
Ice sheet in peril
Taking race out of human genetics
Research
Why do batteries fail?
Hair follicle aging is driven by transepidermal elimination of stem cells via COL17A1 proteolysis
Transmutable nanoparticles with reconfigurable surface ligands
Nematicity in stripe-ordered cuprates probed via resonant x-ray scattering
Transmutable nanoparticles with reconfigurable surface ligands
Diamond family of nanoparticle superlattices
Electrical switching of an antiferromagnet
Holocene deceleration of the Greenland Ice Sheet.
Deformed wing virus is a recent global epidemic in honeybees driven by Varroa mites
Europe’s forest management did not mitigate climate warming
Biophysical climate impacts of recent changes in global forest cover
Allele-specific inhibitors inactivate mutant KRAS G12C by a trapping mechanism
Structures of aminoarabinose transferase AT suggest a molecular basis for lipid A glycosylation
Foxc1 reinforces quiescence in self-renewing hair follicle stem cells
A small-molecule inhibitor of sarcomere contractility suppresses hypertrophic cardiomyopathy in mice